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Molecules | Free Full-Text | Mipsagargin: The Beginning—Not the  End—of Thapsigargin Prodrug-Based Cancer Therapeutics
Molecules | Free Full-Text | Mipsagargin: The Beginning—Not the End—of Thapsigargin Prodrug-Based Cancer Therapeutics

Tunicamycin specifically aggravates ER stress and overcomes chemoresistance  in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation |  Journal of Experimental & Clinical Cancer Research | Full Text
Tunicamycin specifically aggravates ER stress and overcomes chemoresistance in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation | Journal of Experimental & Clinical Cancer Research | Full Text

Proteomic Signatures in Thapsigargin-Treated Hepatoma Cells | Chemical  Research in Toxicology
Proteomic Signatures in Thapsigargin-Treated Hepatoma Cells | Chemical Research in Toxicology

Thapsigargin induces apoptosis when autophagy is inhibited in HepG2 cells  and both processes are regulated by ROS-dependent pathway - ScienceDirect
Thapsigargin induces apoptosis when autophagy is inhibited in HepG2 cells and both processes are regulated by ROS-dependent pathway - ScienceDirect

Pancreatic INS-1 β-Cell Response to Thapsigargin and Rotenone: A  Comparative Proteomics Analysis Uncovers Key Pathways of β-Cell Dysfunction  | Chemical Research in Toxicology
Pancreatic INS-1 β-Cell Response to Thapsigargin and Rotenone: A Comparative Proteomics Analysis Uncovers Key Pathways of β-Cell Dysfunction | Chemical Research in Toxicology

Differential impact of imipramine on thapsigargin- and tunicamycin-induced  endoplasmic reticulum stress and mitochondrial dysfunction in neuroblastoma  SH-SY5Y cells - ScienceDirect
Differential impact of imipramine on thapsigargin- and tunicamycin-induced endoplasmic reticulum stress and mitochondrial dysfunction in neuroblastoma SH-SY5Y cells - ScienceDirect

Thapsigargin promotes colorectal cancer cell migration through upregulation  of lncRNA MALAT1
Thapsigargin promotes colorectal cancer cell migration through upregulation of lncRNA MALAT1

Cell death induced by the ER stressor thapsigargin involves death receptor  5, a non-autophagic function of MAP1LC3B, and distinct contributions from  unfolded protein response components | Cell Communication and Signaling |  Full Text
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text

Endoplasmic reticulum stress signalling – from basic mechanisms to clinical  applications - Almanza - 2019 - The FEBS Journal - Wiley Online Library
Endoplasmic reticulum stress signalling – from basic mechanisms to clinical applications - Almanza - 2019 - The FEBS Journal - Wiley Online Library

Molecules | Free Full-Text | Mipsagargin: The Beginning—Not the  End—of Thapsigargin Prodrug-Based Cancer Therapeutics
Molecules | Free Full-Text | Mipsagargin: The Beginning—Not the End—of Thapsigargin Prodrug-Based Cancer Therapeutics

Knocking out the door to tunicamycin entry | PNAS
Knocking out the door to tunicamycin entry | PNAS

Endoplasmic reticulum stress stimulates the release of extracellular  vesicles carrying danger-associated molecular pattern (DAMP) molecules |  Oncotarget
Endoplasmic reticulum stress stimulates the release of extracellular vesicles carrying danger-associated molecular pattern (DAMP) molecules | Oncotarget

Frontiers | The Cross-Links of Endoplasmic Reticulum Stress, Autophagy, and  Neurodegeneration in Parkinson's Disease
Frontiers | The Cross-Links of Endoplasmic Reticulum Stress, Autophagy, and Neurodegeneration in Parkinson's Disease

Cell death induced by the ER stressor thapsigargin involves death receptor  5, a non-autophagic function of MAP1LC3B, and distinct contributions from  unfolded protein response components | Cell Communication and Signaling |  Full Text
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text

SEC24A identified as an essential mediator of thapsigargin-induced cell  death in a genome-wide CRISPR/Cas9 screen | Cell Death Discovery
SEC24A identified as an essential mediator of thapsigargin-induced cell death in a genome-wide CRISPR/Cas9 screen | Cell Death Discovery

IJMS | Free Full-Text | Thapsigargin—From Traditional Medicine to  Anticancer Drug
IJMS | Free Full-Text | Thapsigargin—From Traditional Medicine to Anticancer Drug

MANF Ablation Causes Prolonged Activation of the UPR without  Neurodegeneration in the Mouse Midbrain Dopamine System | eNeuro
MANF Ablation Causes Prolonged Activation of the UPR without Neurodegeneration in the Mouse Midbrain Dopamine System | eNeuro

Neuronal Apoptosis Induced by Endoplasmic Reticulum Stress Is Regulated by  ATF4–CHOP-Mediated Induction of the Bcl-2 Homology 3-Only Member PUMA |  Journal of Neuroscience
Neuronal Apoptosis Induced by Endoplasmic Reticulum Stress Is Regulated by ATF4–CHOP-Mediated Induction of the Bcl-2 Homology 3-Only Member PUMA | Journal of Neuroscience

Frontiers | STING, the Endoplasmic Reticulum, and Mitochondria: Is Three a  Crowd or a Conversation?
Frontiers | STING, the Endoplasmic Reticulum, and Mitochondria: Is Three a Crowd or a Conversation?

Luteolin prevents liver from tunicamycin-induced endoplasmic reticulum  stress via nuclear factor erythroid 2-related factor 2-dependent sestrin 2  induction - ScienceDirect
Luteolin prevents liver from tunicamycin-induced endoplasmic reticulum stress via nuclear factor erythroid 2-related factor 2-dependent sestrin 2 induction - ScienceDirect

Cells | Free Full-Text | Calcium Pathways in Human Neutrophils—The Extended  Effects of Thapsigargin and ML-9
Cells | Free Full-Text | Calcium Pathways in Human Neutrophils—The Extended Effects of Thapsigargin and ML-9

Molecular mechanism of ER stress‐induced gene expression of tumor necrosis  factor‐related apoptosis‐inducing ligand (TRAIL) in macrophages - Huang -  2015 - The FEBS Journal - Wiley Online Library
Molecular mechanism of ER stress‐induced gene expression of tumor necrosis factor‐related apoptosis‐inducing ligand (TRAIL) in macrophages - Huang - 2015 - The FEBS Journal - Wiley Online Library

Promising therapeutic effect of thapsigargin nanoparticles on chronic  kidney disease through the activation of Nrf2 and FoxO1 | Aging
Promising therapeutic effect of thapsigargin nanoparticles on chronic kidney disease through the activation of Nrf2 and FoxO1 | Aging

IJMS | Free Full-Text | Valproate Attenuates Endoplasmic Reticulum  Stress-Induced Apoptosis in SH-SY5Y Cells via the AKT/GSK3β Signaling  Pathway
IJMS | Free Full-Text | Valproate Attenuates Endoplasmic Reticulum Stress-Induced Apoptosis in SH-SY5Y Cells via the AKT/GSK3β Signaling Pathway

HRC promotes anoikis resistance and metastasis by suppressing endoplasmic  reticulum stress in hepatocellular carcinoma
HRC promotes anoikis resistance and metastasis by suppressing endoplasmic reticulum stress in hepatocellular carcinoma