ER stress-induced cell death mechanisms - ScienceDirect
Tunicamycin specifically aggravates ER stress and overcomes chemoresistance in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation | Journal of Experimental & Clinical Cancer Research | Full Text
Schematic diagram of thapsigargin-stimulated TRAIL-induced apoptosis in... | Download Scientific Diagram
What is Thapsigargin? Frequently Asked Questions
Neuron‐specific translational control shift ensures proteostatic resilience during ER stress | The EMBO Journal
IJMS | Free Full-Text | Thapsigargin—From Traditional Medicine to Anticancer Drug
HRC promotes anoikis resistance and metastasis by suppressing endoplasmic reticulum stress in hepatocellular carcinoma
Thapsigargin - Wikipedia
Unfolded Protein Response (UPR) and Endoplasmic Reticulum (ER) Stress FAQs
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences
JNK activation by thapsigargin versus saturated free fatty acid (SFA).... | Download Scientific Diagram
Thapsigargin promotes colorectal cancer cell migration through upregulation of lncRNA MALAT1
Revealing Protein Aggregates under Thapsigargin-Induced ER Stress Using an ER-Targeted Thioflavin | ACS Sensors
ER stress-induced cell death proceeds independently of the TRAIL-R2 signaling axis in pancreatic β cells | Cell Death Discovery
MITOL prevents ER stress‐induced apoptosis by IRE1α ubiquitylation at ER–mitochondria contact sites | The EMBO Journal
Endoplasmic Reticulum Stress Induces G2 Cell-Cycle Arrest via mRNA Translation of the p53 Isoform p53/47 - ScienceDirect
Pancreatic INS-1 β-Cell Response to Thapsigargin and Rotenone: A Comparative Proteomics Analysis Uncovers Key Pathways of β-Cell Dysfunction | Chemical Research in Toxicology
Endoplasmic reticulum stress signalling – from basic mechanisms to clinical applications - Almanza - 2019 - The FEBS Journal - Wiley Online Library
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text
Phenotypic Discovery of Neuroprotective Agents by Regulation of Tau Proteostasis via Stress‐Responsive Activation of PERK Signaling - Shin - 2021 - Angewandte Chemie International Edition - Wiley Online Library
Endoplasmic reticulum stress, the unfolded protein response and autophagy in kidney diseases | Nature Reviews Nephrology
Targeting endoplasmic reticulum stress in insulin resistance: Trends in Endocrinology & Metabolism
Biomedicines | Free Full-Text | The Crosstalk of Endoplasmic Reticulum (ER) Stress Pathways with NF-κB: Complex Mechanisms Relevant for Cancer, Inflammation and Infection
