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Response to Crizotinib Re-administration After Progression on Lorlatinib in  a Patient With ALK-rearranged Non–small-cell Lung Cancer - Clinical Lung  Cancer
Response to Crizotinib Re-administration After Progression on Lorlatinib in a Patient With ALK-rearranged Non–small-cell Lung Cancer - Clinical Lung Cancer

Responses to the multitargeted MET/ALK/ROS1 inhibitor crizotinib and  co-occurring mutations in lung adenocarcinomas with MET amplification or MET  exon 14 skipping mutation. | Semantic Scholar
Responses to the multitargeted MET/ALK/ROS1 inhibitor crizotinib and co-occurring mutations in lung adenocarcinomas with MET amplification or MET exon 14 skipping mutation. | Semantic Scholar

Downregulation of MET in H1993 cells, effects of crizotinib on EGFR... |  Download Scientific Diagram
Downregulation of MET in H1993 cells, effects of crizotinib on EGFR... | Download Scientific Diagram

Effects of crizotinib on ALK/MET phosphorylation, AKT and ERK... | Download  Scientific Diagram
Effects of crizotinib on ALK/MET phosphorylation, AKT and ERK... | Download Scientific Diagram

Cyclosporine A sensitizes lung cancer cells to crizotinib through  inhibition of the Ca2+/calcineurin/Erk pathway - eBioMedicine
Cyclosporine A sensitizes lung cancer cells to crizotinib through inhibition of the Ca2+/calcineurin/Erk pathway - eBioMedicine

Crizotinib is an Orally Active, ATP-Competitive ALK and c-Met inhibitor -  Network of Cancer Research
Crizotinib is an Orally Active, ATP-Competitive ALK and c-Met inhibitor - Network of Cancer Research

Responses to the multitargeted MET/ALK/ROS1 inhibitor crizotinib and  co-occurring mutations in lung adenocarcinomas with MET amplification or MET  exon 14 skipping mutation - Lung Cancer
Responses to the multitargeted MET/ALK/ROS1 inhibitor crizotinib and co-occurring mutations in lung adenocarcinomas with MET amplification or MET exon 14 skipping mutation - Lung Cancer

Crizotinib hydrochloride | c-Met 阻害剤
Crizotinib hydrochloride | c-Met 阻害剤

MET Tyrosine Kinase Inhibitor Crizotinib (PF-02341066) Shows Differential  Antitumor Effects in Non-small Cell Lung Cancer According to MET  Alterations - Journal of Thoracic Oncology
MET Tyrosine Kinase Inhibitor Crizotinib (PF-02341066) Shows Differential Antitumor Effects in Non-small Cell Lung Cancer According to MET Alterations - Journal of Thoracic Oncology

Chidamide increases the sensitivity of Non-small Cell Lung Cancer to  Crizotinib by decreasing c-MET mRNA methylation [Abstract]
Chidamide increases the sensitivity of Non-small Cell Lung Cancer to Crizotinib by decreasing c-MET mRNA methylation [Abstract]

cに応じられた阻害 | c-Met Inhibition
cに応じられた阻害 | c-Met Inhibition

Antitumor activity of crizotinib in lung cancers harboring a MET exon 14  alteration | Nature Medicine
Antitumor activity of crizotinib in lung cancers harboring a MET exon 14 alteration | Nature Medicine

The promise of selective MET inhibitors in non-small cell lung cancer with  MET exon 14 skipping - Cancer Treatment Reviews
The promise of selective MET inhibitors in non-small cell lung cancer with MET exon 14 skipping - Cancer Treatment Reviews

CRZ in MET-Rearranged NSCLC - Lung Cancer - 2016 American Society of  Clinical Oncology Annual Meeting - Oncology - Clinical Care Options
CRZ in MET-Rearranged NSCLC - Lung Cancer - 2016 American Society of Clinical Oncology Annual Meeting - Oncology - Clinical Care Options

Crizotinib (PF-02341066) | ALK/c-Met/ROS1阻害剤 | MedChemExpress
Crizotinib (PF-02341066) | ALK/c-Met/ROS1阻害剤 | MedChemExpress

PLOS ONE: Ability of the Met Kinase Inhibitor Crizotinib and New Generation  EGFR Inhibitors to Overcome Resistance to EGFR Inhibitors
PLOS ONE: Ability of the Met Kinase Inhibitor Crizotinib and New Generation EGFR Inhibitors to Overcome Resistance to EGFR Inhibitors

Expanding treatment options in NSCLC patients with rare mutations: ALK,  ROS1, MET, BRAF - memoinOncology
Expanding treatment options in NSCLC patients with rare mutations: ALK, ROS1, MET, BRAF - memoinOncology

Crizotinib inhibits activation of MET pathway caused by MET extracellular  SEMA domain duplication - Lung Cancer
Crizotinib inhibits activation of MET pathway caused by MET extracellular SEMA domain duplication - Lung Cancer

Crizotinib in Patients With MET-Amplified NSCLC - Journal of Thoracic  Oncology
Crizotinib in Patients With MET-Amplified NSCLC - Journal of Thoracic Oncology

Characterization of MET exon 14 alteration and association with clinical  outcomes of crizotinib in Chinese lung cancers - Lung Cancer
Characterization of MET exon 14 alteration and association with clinical outcomes of crizotinib in Chinese lung cancers - Lung Cancer

Crizotinib in c-MET- or ROS1-positive NSCLC: results of the AcSé phase II  trial - Annals of Oncology
Crizotinib in c-MET- or ROS1-positive NSCLC: results of the AcSé phase II trial - Annals of Oncology

Response to Crizotinib in a Patient With Lung Adenocarcinoma Harboring a MET  Splice Site Mutation - Clinical Lung Cancer
Response to Crizotinib in a Patient With Lung Adenocarcinoma Harboring a MET Splice Site Mutation - Clinical Lung Cancer

Crizotinib-resistant MET mutations in gastric cancer patients are sensitive  to type II tyrosine kinase inhibitors | Future Oncology
Crizotinib-resistant MET mutations in gastric cancer patients are sensitive to type II tyrosine kinase inhibitors | Future Oncology

Antitumor activity of crizotinib in lung cancers harboring a MET exon 14  alteration | Nature Medicine
Antitumor activity of crizotinib in lung cancers harboring a MET exon 14 alteration | Nature Medicine

Effect of Crizotinib on c-MET expression in c-MET altered cancer cells....  | Download Scientific Diagram
Effect of Crizotinib on c-MET expression in c-MET altered cancer cells.... | Download Scientific Diagram